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At this point, if the muscle remains in a long-term contraction state, the content of ATP generated by oxidative phosphorylation of respiratory chain increases, and electrons overflow into ETC to generate ROS ( 2+ sensitivity of myofibril and damage the contractile function of muscle, possibly through enzyme complexes i and iii (Ci, Ciii)-dependent and/or reduced nicotinamide adenine dinucleotide (NADPH) oxidase-dependent pathways

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