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AMoRe : An automated package for molecular replacement

The globally fitted model correctly captured the more substantial contribution of the RSK to SOS negative feedback strength compared to the positive feedback from ERK to Raf, which resulted in the upregulation of pMEK after inhibition of MEK (Fig

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Signal transducer and activator of transcription 3 (STAT3) binds to DNA response elements in the promoters of ferroptosis-related genes (FTH1, SLC7A11, and GPX4) and regulates their expression

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[3,7,8] The presence of TIS senescent cells in the tumor microenvironment confers resistance to chemotherapy and immunoblockade therapy