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Septic cardiomyopathy (SIC) In a model of LPS-mediated septic cardiomyopathy, some researchers revealed that LPS caused ferroptosis and septic cardiac injury by downregulating GPX4 protein levels and increasing COX-2 [93]

Cyanidin ameliorates endotoxin-induced myocardial toxicity by modulating inflammation and oxidative stress through mitochondria and other factors

One of the most effective ways to bolster your body's defense against oxidative stress and maintain overall wellness is through antioxidants

10 Conclusions and future perspectives In conclusion, studying the mechanisms of comorbidity through the perspective of ferroptosis is essential, with GPX4 and Nrf2 being highlighted as critical targets

Specifically, GOT-1 is involved in the metabolic process of glutamine, where glutamine is transformed into glutamate by the enzyme glutaminase (GLS), and then GOT-1 changes glutamate into alpha-ketoglutarate (-KG) [54, 75]