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In vitro and in vivo laboratory studies have examined its interactions with fibroblasts, tendon cells, and ligament tissue in controlled research models

Zamanian et al., 2020), but its utility still requires a more robust clinical trial to prove

H (400 MHz, MeOD): 7.927.85 (3 H, m, ArH), 7.847.72 (12 H, m, ArH), 4.77 (1 H, dd, J = 9.5, 4.4 Hz, H-4), 4.04 (1 H, t, J = 6.3 Hz, H-1), 3.90 (1 H, d, J = 17.7 Hz, H-6 A or B ), 3.85 (1 H, d, J = 17.7 Hz, H-6 A or B ), 3.483.36 (4 H, m, PCH 2 + H-7), 3.323.25 (1 H, m, H-5 A or B partially obscured by residual peaks in NMR solvent), 3.243.15 (2 H, m, H-8), 2.95 (1 H, dd, J = 13.9, 9.5 Hz, H-5 A or B ), 2.58 (2 H, t, J = 7.1 Hz, H-3), 2.312.11 (2 H, m, H-2), 1.781.65 (2 H, m, CH 2 ), 1.651.52 (4 H, m, CH 2 )

S-GluA1 (Fig

The Australian Regenerative Medicine Institute is supported by grants from the State Government of Victoria and the Australian Government

Antioxidants (Basel) (2021) 10(9):1483