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The study on Sendai in 1997 showed that adsorption of Sendai virus to the cell wall caused a loss of GSH, which affected the Na + /H + antiporter leading to a lower intracellular pH

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Key genes involved in these processes, such as TRMT10C , MTIF3 , and mitochondrial ribosomal protein genes ( MRPSs , MRPLs ), showed increased H3K4me3 signal, suggesting transcriptional activation of the mitochondrial gene expression machinery in MEC-1/N M cells (Table S6)

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