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Of note, the knockdown of vit genes, which encoded vitellogenins, could inhibit the low lipid phenotype of the pry-1 mutant

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However, mutations in Pex3, Pex16, and Pex19 result in the complete absence of peroxisomes and are coupled to the most severe phenotypes (Steinberg et al., 2012)

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10.1016/0006-291X(68)90721-3

Fernandez A, Meechan DW, Karpinski BA, Paronett EM, Bryan CA, Rutz HL, et al
