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Description
Molecular Pathology

Report of fifteen cases

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Hemin is a suppressor of BACH1 DNAbinding activity and induces BACH1's nuclear export and degradation, [ 23 , 27 , 35 , 482 , 483 ] which can reverse the inhibition of VEGF activity in human microvascular endothelial cells (HMEC1) cells caused by hyperoxia by inhibiting BACH1

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