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8 Mice with Gpx4 haploinsufficiency in neutrophils developed spontaneous lupus-like disease, while Gpx4 fl/fl LysMCre + mice exhibited mild autoimmunity

Johnstone TB, Gu Z, Yoshimura RF, Villegier AS, Hogenkamp DJ, Whittemore ER, et al
15 The above suggests that GABAergic system dysregulation plays a significant role in TS pathogenesis and may provide insights into its underlying mechanisms

In addition, Chrysin and inflexin have also been reported as potential agents in neuroinflammation [193, 194]
