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it appears to inhibit the activation of certain inflammatory pathways and cytokines [26]

Depletion of bioavailable nitric oxide in the vasculature by excess oxidants causes endothelial dysfunction, which is characterized by a decrease in endothelium-dependent vasodilation while retaining the ability of vascular smooth muscle cells to vasodilate in response to nitric oxide donors, such as sodium nitroprusside [53]

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Following rotisserie hybridization and incubation for the appropriately assigned time, 500 ul of each sample was added to 500 ul of ice-cold acetonitrile

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