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(MIC 0.1254 g/mL) and potentiates multiple antifungal drugs (Gao et al., 2020)

Lipid transporter protein disrupts TECs lipid uptake, exacerbating lipid metabolism disorders

Considering the consistency and reproducibility of our results across multiple cancer cell lines, we speculated that DSF/Cu might activate cancer cell-intrinsic innate immunity through a shared mechanism

As shown in Figure 5B, BPC-157 promoted the migration of HUVECs in a concentration-dependent manner

Copper is an essential cofactor for superoxide dismutase (SOD), a primary antioxidant enzyme, and GHK-Cu has been studied in models examining oxidative stress mitigation

Belaidi, A