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however, OCTN1 protein expression remained comparable, proposing that the oxidative stress in DMD imposed peroxidative damage on mitochondrial membrane, then degrading OCTN1 [90]
An emerging experimental approach involves the direct transfer of healthy donor mitochondria into CAR-T cells [256, 257]
S.ChauhanP
297 Mtb exploits DosRST to establish and maintain nonreplicating persistence in response to hypoxia, NO, acid stress, or starvation
Oxidation of hydrogen sulfide by human liver mitochondria
As with embryos exposed in vivo, the concentration range for teratogenicity was very narrow (Slott & Hales, 1986)
