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By contrast, the intranuclear amount of p65 of NF-B, which can suppress the gene expression of CYP1A2, CYP2E1, and CYP3A11, was higher in untreated IL-1ra KO than WT mice

This review provides a foundational understanding for the development of targeted treatments involving SLC7A11 for managing fibrotic diseases.

While more research is needed, preliminary studies suggest potential benefits in managing symptoms and improving quality of life

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