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doi: 10.1016/j.bbalip.2008.06.005

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PTMs such as phosphorylation and ubiquitination control the activity, stability, and interactions of lipid-metabolizing enzymes, enabling immune cells to rapidly adjust their metabolic pathways in response to immune activation

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Both work through separate biological mechanisms: semaglutide acts on GLP-1 receptors to reduce appetite and improve glucose control, while L-carnitine supports energy substrate utilization at the cellular level