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Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing sp1 glycosylation

GDE-treated cells exhibited upregulation of N1 markers, along with increased NETs formation, ROS production, and proinflammatory cytokine secretion, all of which were significantly suppressed by L-BSO treatment (Fig

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Over time, I have dropped in my own great loves to stand in her story, and it makes me cry for both of us what we lost, what we learned and how we survived