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Furthermore, oxidative and excitotoxic damage to glial cells within the median eminence and organum vasculosum of the lamina terminalis (OVLT), key circumventricular organs lacking a complete bloodbrain barrier, may impair the transduction of peripheral osmotic signals to neuroendocrine outputs

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The mitochondrial permeability transition pore: molecular nature and role as a target in cardioprotection

Acta-Protein Struct

Previous studies have shown that AEC could differentiate into fibroblasts/myofibroblasts increasing fibrotic tissue deposition and that EMT contributes to fibroblast accumulation and consequent development of pulmonary fibrosis in animal models [245,246,247,248]