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The study concluded that the loss of SCFAs contributes to an imbalance in pro- and anti-inflammatory pathways in the CNS, exacerbating neuroinflammation in CKD (Vaziri et al., 2013)
The main mechanisms with research support: Collagen and connective tissue: GHK-Cu upregulates collagen synthesis and metalloproteinases the enzymes that remodel old, damaged collagen
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