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Both are worth investigating, though for different reasons

As mentioned before, a critical limitation of these strategies lies in the degeneracy of dominant nociceptor subtypes that may dynamically shift following nerve injury or over time, diminishing the efficacy of highly selective monotherapies [361, 362]

Cellular Energy and Fatigue By improving mitochondrial efficiency and ATP generation, MOTS-c supports energy balance under metabolic or physical stress, helping reduce fatigue and improve vitality

Therefore, the EORS hypothesis of aging consists of the inability to respond to energy demands or stress that leads to stress-induced catastrophic initiation of cellular death pathways and brain function decline

The peptide reduced motor disability, preserved myelin integrity, and supported neurological function in these experimental paradigms

The timeline for noticeable improvements can vary depending on factors such as wound severity, overall health, and treatment consistency