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Similar metabolic effects were observed with extended treatment

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Because the [ 13 C 1 ]acetate tracer enters the acetyl-coenzyme A pool and is oxidized to 13 CO 2 in the Krebs cycle, we measured recovery of 13 CO 2 after an infusion of [ 13 C 1 ]acetate (as an index of Krebs cycle function) and found no change with GSH improvement, suggesting that Krebs cycle function was not altered and that the defect must lie before it

doi: 10.1111/exd.12339

75,76 Although other sources of oxidants (H 2 O 2 ) than the electron transport chain have been identified in relation to fatty acid metabolism within mitochondria, 77,78 the formation of peroxynitrite as the critical oxidant for mitochondrial dysfunction requires superoxide, which is generated by the electron transport chain