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with IGF-1 levels remaining elevated with a single oral dose for up to 24 hours [R]

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Notably, the absence of LAT1 impedes the differentiation pathways of Th1, Th2, and Th17 cells, acting independently of T cell receptor signals but via the PI3K/Akt/mTOR pathway

Recent studies have suggested that mitochondrial dysfunction contributes to the pathophysiology of NMOSD (136, 137)

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