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Back to Archived Journals Research and Reports in Neonatology Volume 6 Screening for medium-chain acyl CoA dehydrogenase deficiency: current perspectives Authors Soler-Alfonso C, Bennett M, Ficicioglu C Received 3 June 2015 Accepted for publication 16 September 2015 Published 18 January 2016 Volume 2016:6 Pages 110 DOI Checked for plagiarism Yes Review by Single anonymous peer review Peer reviewer comments 5 Editor who approved publication: Dr Robert Schelonka Claudia Soler-Alfonso, 1 Michael J Bennett, 2 Can Ficicioglu 1 1 Department of Pediatrics, Section of Metabolic Disease, 2 Department of Pathology and Laboratory Medicine, The Childrens Hospital of Philadelphia and Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA Abstract: Medium-chain acyl-CoA dehydrogenase (MCAD) deficiency is the most common disorder associated with fatty acid oxidation

On the contrary, other studies have reported a plethora of autoantibodies in LC, including elevated ANA antibodies (e.g., anti-U1-snRNP and anti-SS-B/La), antineuronal antibodies, anti-IFN antibodies, autoantibodies against ACE2, the angiotensin II AT 1 receptor, the angiotensin 17 MAS receptor, 2 -the adrenoreceptor and the muscarinic M2 receptor [146,190,194,195,197,247]

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ROS are also responsible for disrupting mitochondrial calcium homeostasis, a process mediated by voltage-dependent anion channels and the mitochondrial calcium uniporter [168]