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Description

In the first mechanism, HHI-induced Smoothened inhibition activates noncanonical Hedgehog signaling pathways, which enhances calcium influx into myocytes and increases muscle contractility [10, 110, 111]

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Very-long-chain acyl-CoA dehydrogenase deficiency (VLCADD) Very-long-chain acyl-CoA dehydrogenase deficiency (VLCADD), like MCAD deficiency, affects the breakdown of fatty acids

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While the functional effect of acetylation/deacetylation has been shown for only three mitochondrial proteins, this mechanism of regulation has tremendous potential for linking the altered mitochondrial proteome with mitochondrial function