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However, even in one type of cancers, e.g., NSCLC, the impact of c-MET copy number on prognosis is a histological subtype dependent: c-MET amplification with a poorer prognosis in patients with adenocarcinoma but not in those with squamous cell carcinoma [21, 22]

doi:10.2174/1389201021666200320135849 Gargouri B, Carstensen J, Bhatia HS, Huell M, Dietz GPH, Fiebich BL

This suggests an important regulatory role of these Cys, including the potential for modification via S -glutathionylation

10.1161/CIRCRESAHA.114.300559 [DOI] [PMC free article] [PubMed] [Google Scholar] 283.Gao, M

Nature 6787 , 701704, (2000)

While AdoMet is the main methyl donor, there are three methyltransferases that are not AdoMet-dependent: Cdc21, Met6, and Mgt1