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Dietary interventions provide a non-pharmacological means to influence tumor and immune metabolism, and ketogenic diets, by limiting glucose availability and increasing ketone bodies, may impair tumor glycolysis while supporting Teff oxidative metabolism [130, 131]

observed an upregulation of Wnt3a, -catenin, VEGF, and Cyclin D1, as well as a downregulation of GSK-3 and caspase 3 in a rat model of MCAO following isoflurane injection

Figure 1 Similarly, MnO 2 NPs can react with GSH in tumor cells to generate GSSH and Mn 2+ , which would result in the in uncontrolled reactive oxygen species (ROS) levels in tumor cells due to the decreased GSH level ( Figure 2 )

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Ni (n=2) only in prokaryotes

NAC treatment began at 3 weeks of age, and all mice were sampled at 9 weeks of age