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Senolytic peptide FOXO4-DRI: published animal dosing vs community-derived structure, 10 mg/3 mL reconstitution math, safety, and evidence boundaries.
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Aims/hypothesis Barth syndrome is a mitochondrial disorder caused by Tafazzin (TAZ) mutations, which impair cardiolipin remodelling and contribute to systemic metabolic alterations
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Because these redox changes were seen in the cytosol rather than mitochondria, we next explored the thiol network in the context of PRKN expression
Rizk et al
