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Eugenol has been shown to downregulate PGC-1 expression and reduce mitochondrial membrane potential in AML cells, inhibiting their growth and promoting apoptosis through the mitochondrial pathway in AML patients [147]
Wallerian degeneration and peripheral nerve conditions for both axonal regeneration and neuropathic pain induction
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Additionally, Cu disrupts the mitochondrial membrane potential, ultimately culminating in apoptotic cell death
