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In general, when treating KRAS-driven tumours, it is necessary to pay attention to other gene changes and pay attention to individualised treatment (Fig

Genetica 142 , 495505 (2014)

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Both examples reinforce the view that other mechanisms beyond solely triplication of Hsa21 are involved to explain the wide phenotypic variation occurring in DS individuals

In order to decrease the model size and improve the computational efficiency, only amino acids 1941532 (UniProtKB-Q8CG09) were included into the structure generation process, disregarding the N-terminal transmembrane domain (TMD) 0