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The rationale is complementary mechanisms: TB-500 drives cell migration to injury sites through actin regulation, while BPC-157 supports blood vessel formation, growth factor expression, and anti-inflammatory signalling through nitric oxide pathway modulation

Lee et al., 2016a

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At E15, they are predominantly expressed within the midbrain, medulla and spinal cord, with expression in the latter two diminishing after E16 and expression within the hypothalamus and nucleus accumbens subsequently increasing