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Its a peptide too large and too polar to cross the gut, and it gets destroyed by enzymes before itd get the chance

Since the 1960s it has been known that NAG is an enzyme present in kidney tissues,38 in the lysosomes of the proximal tubular cells, and that its excretion in urine increases when cells are damaged and NAG is releases into the tubular lumen .39 Classically, the determination of NAG was used to evaluate the nephrotoxicity caused by aminoglycoside antibiotics.40 Moreover, NAG levels may be increased in cases of elevated intratubular pressure as in urinary tract obstruction41 and in VUR, although in this case, as occurs with albuminuria, it is difficult to know if it is due to the elevated pressure or to the associated reduction of glomerular filtration.36 On basic determinations of kidney function as markers of parenchyma loss In a study including 77 children with normal renal parenchyma as confirmed by dimercaptosuccinic acid scan and 102 children with loss of parenchyma (one or more scars, single kidney, hypodysplasia), we determined the quality and diagnostic efficiency of the kidney function markers listed above

S2CID 234472935

Years of alcohol abuse followed by heavy cannabis usage during withdrawal recovery had messed up his body

The quantification standard curve was obtained ranging from 6 nM to 20 M

This lack of clinical evidence is the primary reason why these compounds are not currently part of standard medical practice