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A putative mechanism of age-related synaptic dysfunction based on the impact of IGF-1 receptor signaling on synaptic CaMKII phosphorylation

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Momentum, exactly

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The loss of Regucalcin in Nematoda, further suggests that this gene does not perform other essential functions unrelated to VC synthesis, or alternatively, that this gene might have been functionally replaced by others in this group of species

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