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E., Jensen, L

Conversely, attempting to stimulate Phase 2 pathways without sufficient Phase 1 activity can impair the bodys ability to process fat-soluble toxins, limiting overall detoxification efficiency and allowing toxic burden to persist

Several repurposed diabetes drugs and natural compounds have been shown to exert neuroprotective effects by inhibiting ferroptosis

Finally, two synthetic ITC protected dopaminergic neurons and prevented motor deficits associated with PD in mice (88, 89)

Glutathione injections may support these treatments by helping manage oxidative stress during the repair phase
