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In part, the bimodal effects of NO may relate to differences between its multiple mammalian synthetic enzymes, with neuronal nNOS and vascular eNOS tending to be more neuroprotective, while inflammation-associated glial and immune iNOS activity tends to be more neurotoxic (Puzzo et al., 2006

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10.1038/s41419-020-2298-2 72 LiM.WangQ.LiY.CaoS.ZhangY.WangZ.et al (2020b)