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As a result, the ETC functioning is disrupted, and ATP content becomes significantly depleted, which breaks down metabolic and ionic homeostasis and activates the degradative enzymes such as nucleases, proteases, and phospholipases [182, 188]

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The protective effects of carnosine and related compounds against the oxidative damage of human SOD by peroxyl radicals generated from 2,2-azobis(2-amidinopropane) dihydrochloride (AAPH) were established