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Further investigation is needed to reveal whether skin microbes contribute to this process via altered community composition and dysregulated interactions with surrounding epidermal cells (Fig

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Conversely, high oxidative stress can accelerate NAD+ degradation via the enzyme CD38, which consumes NAD+ as part of the inflammatory response

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Toll-like receptor 4 deficiency ameliorates propofol-induced impairments of cognitive function and synaptic plasticity in young mice

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