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10.1007/BF00714275

doi: 10.1158/2326-6066.Cir-20-0384

To place these microbial changes in a broader context, it is useful to compare them with those in inflammatory bowel disease (IBD)

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Current research suggests that RIG-I also has a direct antiviral effect, as evidenced by the greater extent of virus replication in RIG-I-deficient cells than in cells lacking IFNAR1

The position of the GSH-conjugate LTC4 was examined for bovine Mrp1 via cryo-EM, identifying its GS-moiety binding to the P-pocket via hydrogen bonds with both of its carboxyl groups [12]