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The lack of large, well-controlled RCTs limits the clinical application of oxidative stress modulation in FM

Disagreements were resolved by discussion or consensus with a third independent author (CYZ)

Amores-Sanchez MI, Medina MA

Our results indicate increased DNA damage and reduced DNA repair are key targets of the dual agent treatment, and together these suggest the cancer cells are unable to repair increased DNA damage leading to higher cell death, further supporting the potential use of this dual agent as TNBC therapeutic

elegans depend on daf-12/daf-16

Endorphins, a different set of neurotransmitters, are released during vigorous exercise, excitement, and in response to pain, functioning as natural analgesics and mood boosters [95]