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GHK-Cus mechanism depends on cumulative tissue signaling rather than peak concentration

Molecules 26(8):2284

Detrimental effects of ROS on lung tissue include direct damage to the membrane of lung parenchymal cells (lipid damage), modifications of enzymes and proteins important for cell metabolism, and mutations in DNA, resulting in apoptosis of these cells

It plays a role as an antioxidant and supports the breakdown of fat molecules

At low concentrations or short durations of exposure, ozone, as an oxidative preconditioning, effectively enhances the antioxidant capacity with the help of H 2 O 2 , aldehydes, and other products.18 However, with a high concentration of exposure, the amount of ozone exceeds the buffering capacity of the blood, which directly damages cells and results in the production of many inflammatory factors and oxidative damage in a short period through its products.85,86 The acute damage can be respiratory, cardiovascular, or even fatal.124 The accumulation of H 2 O 2 and aldehydes produced by ozone at small dosages over extended periods of exposure can also cause injury