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These findings are mechanistically coherent with GHK-Cus anti-inflammatory NF-kB activity because the secondary injury cascade in SCI involves microglial activation, sustained pro-inflammatory cytokine release, and reactive oxygen species generation

1997 Mitochondrial control of apoptosis Immunol

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Furthermore, ammonium alters the types of cytokines secreted by dendritic cells, diminishing their ability to activate T cells and further limiting anti-tumor immune responses (144)