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Experimental evidence further suggests the alcohol-induced activation of ABL2 promotes ALD by upregulating HIF-1 and subsequent PPAR expression, suggesting that inhibiting ABL2 could be a promising therapeutic target for ALD treatment (Malnassy et al., 2023)

10.1152/physrev.00022.2006 32 HankeT.WongJ

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Moreover, NMDAR currents are upregulated by Fyn kinase, a member of the Src family of non-receptor tyrosine kinases, in human iPSC-derived neurons, as in the neurons from the rodent CNS [32, 115]

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