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Modulate nitric oxide (NO) pathways , theoretically improving microvascular blood flow and reducing inflammation
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TB-500 operates systemically as a **cytoskeleton remodeler**: its primary function is G-actin monomer sequestration (active N-terminal Ac-Ser-Asp-Lys-Pro), its cleaved AcSDKP sequence is an **autonomous angiogenic factor**, it upregulates laminin-5 and matrix metalloproteinases (MMPs), and mobilizes mesenchymal progenitor cells
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Clinicians therefore combine general wound care principles and nursing experience to form practical advice
