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This disruption is characterised by increased permeability evidenced by tracer extravasation, blood product accumulation, and focal tight junction widening and is accompanied by sustained inflammatory signalling, likely driven by ongoing extracellular matrix remodelling and perivascular dysfunction

doi: 10.1007/s00467-005-2085-4

A study comparing various modalities of KRT in eight patients with hyperammonaemia showed that PD was less efficient than intermittent HD or CKRT in reducing ammonia levels 34

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