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Since neurons and skeletal muscle express little ME1, they may be more susceptible to aging-induced cytoplasmic redox imbalance than liver or astrocytes that express higher levels (Supplementary Table 1

Lower levels of GSH were also observed in the substantia nigra of PD patients and the caudate nucleus of progressive supranuclear palsy (PSP) patients 16

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These findings highlight FN-CNPs as a promising and well-tolerated nanotherapeutic for OA intervention (193)

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542 Although tau protein aggregation and beta-amyloid protein (A) accumulation play central roles in the pathophysiological hypothesis of AD, the potential role of metal overload, subsequent oxidative stress, and tissue damage cannot be ignored