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This was first explored by Balthasar and colleagues, who in 2005 proved that specific inhibition of the IgG:FcRn interaction by use of a murine mAb (mouse IgG1, clone 4C9) with V regions specific for rat 2m generated by Bjorkman and colleagues could deplete endogenous IgG levels in rats [113, 114]
doi:10.1161/STROKEAHA.107.504498
"Pharmacokinetics of high doses of cyanocobalamin administered by intravenous injection for 26 weeks in rats"
Additionally, research published in Drug Testing and Analysis characterizing AOD9604 metabolism and confirming its C-terminal HGH fragment structure, supporting its classification as a non-IGF-1-stimulating lipolytic agent has been used by clinicians evaluating the compound for metabolic applications
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