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CRP primarily exerts its effects through binding to Fc receptors on immune cells, initiating downstream signaling pathways, particularly the NF-B pathway (124)
Additionally, CuSAE also exhibits peroxidase (POD)- and glutathione oxidase (GSHOx)-mimicking activities, which elevate oxidative stress in tumor cells, thereby blocking the compensatory repair mechanisms and continuously activating tumor disulfidptosis
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