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This review summarizes current knowledge of the enzymes, structural principles, and cellular mechanisms governing tubulin glutamylation, highlights its emerging roles in human diseases, and discusses new technological advancesincluding biochemical reconstitution, super-resolution imaging, and live-cell manipulation toolsthat are beginning to reveal how this modification dynamically controls microtubule properties and the functions of flagella, cilia, and centrosomes in health and disease

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Under pathological conditions, loss of ATP7B function causes increased autophagic structures in the cytoplasm, positively correlated with cytoplasmic Cu levels

Alkazemi, D.U.Z., Rahman, A., and Habra B., Alterations in glutathione redox homeostasis among adolescents with obesity and anemia, Sci

Nrf2 has also become a consensus in the regulation of neurodegenerative diseases

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