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doi: 10.1016/j.isci.2021.103220
N -acetyl-cysteine (NAC), a glutathione precursor, was administered to patients to determine whether increased levels of brain glutathione would improve MMN and by extension NMDA function
& Reiss, K
Therefore, activating GPX4 and GSH to inhibit ferroptosis is an attractive option for cardiac disease therapy (Fig
Henry MC, Assi SB, Rogier C, Dossou-Yovo J, Chandre F, Guillet P, et al
10.1021/bi401719r 39
