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When the affinity-enhancing mutations were incorporated into a full-length thirty-seven amino acid rat amylin peptide, the potency of rat amylin for calcitonin and amylin receptor 1/2 activation was significantly enhanced by up to 10-fold (Figure 9 and Table 3)

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Coming off the peptides doesn't reverse the repair that occurred

These factors may result in subtherapeutic plasma concentrations and reduced bioavailability [67]