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Researchers observed improvements in corneal nerve fiber density and patient-reported outcome measures following a 28-day treatment period, providing early clinical evidence for the peptides activity at the innate repair receptor [1]

Additionally, TSA improved T cell function by reducing serum levels of high-mobility group box 1 (HMGB1), enhancing macrophage phagocytic activity and promoting bacterial clearance (67)

When compared with the control, in both the studies authors found a significant decrease in GSH upon methylglyoxal treatment

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Unresolved inflammation results in formation of vulnerable plaques characterized by enhanced macrophage apoptosis and defective efferocytosis of apoptotic cells resulting in necrotic cell death leading to increased smooth muscle cell death, decreased extracellular matrix production, and collagen degradation by macrophage proteases