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It activates genomic pathways that produce neuroprotective proteins, shielding neurons from oxidative stress, inflammation, and other neurotoxic factors, thereby preserving brain health
EX-527 treatment inhibited the effect of VB 12 on the improvement of m (Figures 5(e) and 5(f)) and ATP (Figure 5(g)) and downregulated the expression of parkin and increased the expression of LC3II and P62 (Figure 5(h)), similar to NaT treatment
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Figure 2A) and serum triglyceride levels ( p = 0.043
The powder should dissolve on its own
