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Description
We hypothesized that the defects in cell division and actin-based motility would hamper R

They achieve different endpoints

OCallaghan, N

Furthermore, Mdivi-1 has been observed to reduce neuronal damage by decreasing oxidative stress and depolarizing mitochondrial membranes through a mechanism that is independent of Drp1 [129]

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Another study published in 2024 by Vleminckx et al
