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In summary, our results provide clear evidence for the role of C32E8.9 in lifespan extension resulting from impaired mitochondrial translation by modulating innate immunity and lipid reprogramming

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Biosynthesis of riboflavin in Archaea 6,7-dimethyl-8-ribityllumazine synthase of Methanococcus jannaschii

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The surplus of ROS in tissues then attack membrane lipids and proteins and lead to the generation of aldehydic substance malondialdehyde (MDA) and protein-bound carbonyl groups, which are the principal decomposition products respectively for lipid peroxidation and oxidative modification of proteins 13